Petaloid Seborrheic Dermatitis
Written by Jeffrey J. Meffert, LT Col., USAF, MC, Wilford Hall Medical Center // Brooke Army Medical Center // San Antonio, TX 78259 [email protected]
Seborrheic dermatitis skin lesions manifest as branny or greasy scaling over red, inflamed skin. Hypopigmentation is seen in blacks. Infectious eczematoid dermatitis, with oozing and crusting, suggests secondary infection. A seborrheic blepharitis may occur independently.
Distribution follows the oily and hair-bearing areas of the head and the neck, such as the scalp, the forehead, the eyebrows, the lash line, the nasolabial folds, the beard, and the postauricular skin. An extension to submental skin can occur. Presternal or interscapular involvement is more common than nonscaling intertrigo of the umbilicus, axillae, inframammary and inguinal folds, perineum, or anogenital crease, which also may be present.
Two distinct truncal patterns of seborrheic dermatitis can occasionally occur. An annular or geographic petaloid scaling (petaloid seborrheic dermatitis) is the most common. A rare pityriasiform variety can be seen on the trunk and the neck, with peripheral scaling around ovoid patches, mimicking pityriasis rosea.
“Petaloid seborrheic dermatitis” is a frequent presentation of seborrheic dermatitis in people with dark skin. Typically, patients present with red, scaly plaques in the eyebrows and along the melo-labial fold; however, many patients with dark skin present with polycyclic coalescing rings. These rings may be slightly pink or hypopigmented in color and usually do not show significant scale until the area is scraped for a potassium hydroxide (KOH) preparation. The etiology of seborrheic dermatitis is not clear and has been associated with the yeast Pityrosporum orbiculare, the mite Demodex folliculorum, various bacterial colonizations and skin response to the environment, such as changes in temperature, humidity and bath water.
Treatment usually includes the application of a topical corticosteroid containing 1 percent hydrocortisone cream. In refractory cases, 0.2 percent hydrocortisone valerate or 0.5 percent desonide creams may be applied twice daily. Use of stronger corticosteroids on areas other than the scalp is discouraged as it may lead to atrophy, telangiectases, perioral dermatitis and, in people with dark skin, noticeable hypopigmentation. Topical and systemic anti-fungal agents have occasionally been used with good results. Shampoo containing ketoconazole has helped many patients with seborrheic dermatitis of the scalp. Washing the hair and the face with prescription and over-the-counter dandruff shampoos containing selenium sulfide may also help, although washing the face with selenium sulfide more often than two or three times per week can be irritating.
The differential diagnosis for patients with seborrheic dermatitis is broad and may require biopsy to confirm the clinical presentation, especially when usual treatment measures do not work. Sarcoidosis can initially look like seborrheic dermatitis, although confluence along the scalp line and eyebrows is more typical of seborrheic dermatitis. Patients with sarcoidosis may respond to topical corticosteroids but usually require systemic therapy or very potent topical agents to clear the lesions.
Secondary syphilis often presents with rings similar to those pictured here, although the presence of lesions only on the face and not on the palms or soles would be most unusual. A negative serology test will exclude lues in more questionable cases. Tinea faciei may look identical to seborrheic dermatitis, but results of the KOH preparation should be positive and the patient should respond to topical antifungal medications. Discoid lupus erythematosus may start with flesh-colored rings, but hypopigmented, red papules and plaques would be a more common presentation. Diagnosis of discoid lupus is best determined using biopsy, since anti-nuclear antibody serologies are usually negative. Cutaneous T-cell lymphoma (called mycosis fungoides) may also include hypopigmentation; this is a relatively common presentation in young, dark-skinned patients. Again, biopsy for histopathology is necessary to confirm or exclude the diagnosis.
References
- McLaurin CI. Annular facial dermatoses in blacks. Cutis. 1983;32:369–70384.
- Sei Y, Hamaguchi T, Ninomiya J, Nakabayashi A, Takiuchi I. Seborrheic dermatitis: treatment with anti-mycotic agents. J Dermatol. 1994;21:334–40.
Contributing editor is MARC S. BERGER, M.D., C.M., The Reading Hospital and Medical Center, Reading, Pennsylvania.
- Occupational Dermatitis: Caused In The Workplace
- Eczema Nummular Treatment
- Plaque Psoriasis Treatment
- Seborrheic Dermatitis: Spreads To The Face & Beyond
- What factors can aggravate atopic dermatitis?
- Venostasis Dermatitis
- How to Cure Psoriasis
- Lichenoid Dermatitis Information
- Foot Dermatitis Risk Factors
- Molluscum Dermatitis
- Contact Dermatitis
- Dermatitis Herpetiformis Symptoms
- Rhus Dermatitis
- Venous Stasis Dermatitis Treatment
- How do you get Eczema?
- Pyotraumatic Dermatitis
- Risk Factors of Neurodermatitis
- Psoriatic Arthritis Treatment
- Foot Dermatitis Symtoms
- What is Dermatitis?